CoQ10 Supplementation May Help Improve Symptoms of Type-2 Diabetes
Previous research has shown that increased oxidative stress and impaired antioxidant defense contribute to the progression of type-2 diabetes. Additionally, diabetic patients have been shown to have reduced levels of coenzyme Q10 (CoQ10).
Based on this knowledge, researchers from Tehran University of Medical Sciences in Iran set out to determine the effect of CoQ10 supplementation on the glycemic control and lipid profiles of type-2 diabetics. They found that the supplement improved glycemic control and total and LDL cholesterol levels, but had no effects on triglycerides or HDL cholesterol.
Participants in the study included 64 type-2 diabetics who were given either 200 mg CoQ10 or a placebo daily over the course of 12 weeks. The researchers took fasting blood samples and also measured levels of fasting plasma glucose, HbA1c, total cholesterol, triglycerides, LDL cholesterol, and HDL cholesterol.
In addition to the effects on glycemic control and cholesterol mentioned above, the researchers also noted lower levels of HbA1c. HbA1c is a form of hemoglobin that is used to measure average blood glucose levels over time.
These results suggest that type-2 diabetics may be able to improve glycemic control and cholesterol levels by taking supplements of CoQ10.
The study was published in the June 2013 issues of the journal Minerva Gastroenterologica e Dietologica.
CoQ10 is a powerful antioxidant that fuels the pumping of blood to and from the heart, and protects cells from being damaged and destroyed. Previous studies have linked CoQ10 with improved cognitive health and reversing the effects of aging.
After the age of 20 our bodies become less capable of naturally producing CoQ10. If you want to increase your CoQ10 intake, try adding oily fish, organ meats such as liver and hearts, and whole grains to your diet. You might also consider a high quality, high potency supplement. Be sure to choose a supplement that is made with ubiquinol rather than ubiquinone.